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PubMed

Understanding the Notch Signaling Pathway in Acute Myeloid Leukemia Stem Cells: From Hematopoiesis to Neoplasia

Cancers (Basel)
Multidisciplinary Digital Publishing Institute (MDPI), 2022DOI: 10.3390/cancers14061459https://creativecommons.org/licenses/by/4.0/
SIMPLE SUMMARY: We review the state of the art of knowledge regarding the Notch signaling pathway to shed light on the role that this pathway has in hematopoiesis and hematological neoplasia, focusing on acute myeloid leukemia. ABSTRACT: The Notch si...
PubMed

Comparison of TP53 mutations in myelodysplasia and acute leukemia suggests divergent roles in initiation and progression

Blood Neoplasia
The American Society of Hematology, 2024DOI: 10.1016/j.bneo.2024.100004https://creativecommons.org/licenses/by-nc-nd/4.0/
TP53 mutation predicts adverse prognosis in many cancers, including myeloid neoplasms, but the mechanisms by which specific mutations affect disease biology, and whether they differ between disease categories, remain unknown. We analyzed TP53 mutatio...
PubMed

GATA2 deficiency and related myeloid neoplasms

Semin Hematol
2017DOI: 10.1053/j.seminhematol.2017.05.002http://creativecommons.org/licenses/by-nc-nd/4.0/
The GATA2 gene codes for a hematopoietic transcription factor that through its two zinc fingers (ZF) can occupy GATA-DNA motifs in a countless number of genes. It is crucial for the proliferation and maintenance of hematopoietic stem cells. During th...
PubMed

Clonal hematopoiesis and hematological malignancy

J Clin Invest
American Society for Clinical Investigation, 2024DOI: 10.1172/JCI180065https://creativecommons.org/licenses/by/4.0/
Clonal hematopoiesis (CH), the expansion of hematopoietic stem cells and their progeny driven by somatic mutations in leukemia-associated genes, is a common phenomenon that rises in prevalence with advancing age to affect most people older than 70 ye...
PubMed

How I diagnose myeloid neoplasms with germline predisposition

Am J Clin Pathol
Oxford University Press, 2023DOI: 10.1093/ajcp/aqad075© American Society for Clinical Pathology, 2023. This work is written by (a) US Government employee(s) and is in the public domain in the US.
OBJECTIVES: Pathologists play a crucial role in the initial diagnosis of germline predisposition to myeloid neoplasia and subsequent surveillance for disease progression. The diagnostic workup can be challenging, particularly if clinical history, lab...
PubMed

Induction of Chromosomal Instability via Telomere Dysfunction and Epigenetic Alterations in Myeloid Neoplasia

Cancers (Basel)
Multidisciplinary Digital Publishing Institute (MDPI), 2013DOI: 10.3390/cancers5030857https://creativecommons.org/licenses/by/3.0/
Chromosomal instability (CIN) is a characteristic feature of cancer. In this review, we concentrate on mechanisms leading to CIN in myeloid neoplasia, i.e., myelodysplastic syndrome (MDS) or acute myeloid leukemia (AML). The pathogenesis of myeloid n...
PubMed

Individual Telomere Lengths in Chronic Myeloid Leukemia

Neoplasia
Neoplasia Press, 2009DOI: 10.1593/neo.09836Copyright © 2009 Neoplasia Press, Inc. All rights reserved
Chronic myeloid leukemia (CML) is a neoplasia characterized by proliferation of a myeloid cell lineage and chromosome translocation t(9;22) (q34;q11.2). As in the case of most cancers, the average telomere length in CML cells is shorter than that in ...
PubMed

Genomic alterations in patients with somatic loss of the Y chromosome as the sole cytogenetic finding in bone marrow cells

Haematologica
Ferrata Storti Foundation, 2020DOI: 10.3324/haematol.2019.240689https://creativecommons.org/licenses/by-nc/4.0/
Loss of the Y chromosome (LOY) is one of the most common somatic genomic alterations in hematopoietic cells in men. However, due to the high prevalence of LOY as the sole cytogenetic finding in the healthy older population, differentiating isolated L...
PubMed

DDX41-related myeloid neoplasia

Semin Hematol
While early presentation of familial leukemia syndromes is typical, long disease anticipation may mask cases of familial traits in seemingly spontaneous disease. Germline mutations in DDX41 gene have been discovered in several leukemia families, as w...
PubMed

Philadelphia chromosome positive AML arising from JAK2-positive myelofibrosis

Biomark Res
BMC, 2018DOI: 10.1186/s40364-018-0147-6http://creativecommons.org/publicdomain/zero/1.0/
BACKGROUND: A feature of myeloproliferative neoplasia is transforming to more aggressive and malignant myeloid neoplasia, including acute myeloid leukemia. Different pathogenesis mechanisms participate in transformation, including transformation of e...
PubMed

Cohesin-dependent regulation of gene expression during differentiation is lost in cohesin-mutated myeloid malignancies

Blood
The American Society of Hematology, 2019DOI: 10.1182/blood.2019001553© 2019 by The American Society of Hematology
Cohesin complex disruption alters gene expression, and cohesin mutations are common in myeloid neoplasia, suggesting a critical role in hematopoiesis. Here, we explore cohesin dynamics and regulation of hematopoietic stem cell homeostasis and differe...
PubMed

STAT signaling in the pathogenesis and therapy of acute myeloid leukemia and myelodysplastic syndromes

Neoplasia
Neoplasia Press, 2025DOI: 10.1016/j.neo.2025.101137https://creativecommons.org/licenses/by-nc/4.0/
Acute myeloid leukemia (AML) and myelodysplastic syndromes (MDS) represent complex hematopoietic malignancies characterized by ineffective hematopoiesis and dysregulated myeloid differentiation. Recent research has underscored the critical role of ab...
PubMed

Antigens expressed by human B lymphocytes and myeloid stem cells

J Exp Med
The Rockefeller University Press, 1977DOI: 10.1084/jem.146.4.1143http://creativecommons.org/licenses/by-nc-sa/4.0/
Antisera prepared against papain-digested spleen cell membranes were known to by cytotoxic for normal and neoplastic human B lymphocytes and for a majority of acute and chronic myeloid leukemic cells. It is now shown that these antisera are also cyto...
PubMed

Tumor-associated myeloid cells support murine T-ALL in the central nervous system via integrin signaling

Blood Neoplasia
The American Society of Hematology, 2025DOI: 10.1016/j.bneo.2025.100095https://creativecommons.org/licenses/by-nc-nd/4.0/
T-cell acute lymphoblastic leukemia (T-ALL) is a common pediatric malignancy, and central nervous system (CNS) involvement is associated with inferior prognoses. Here, we show that in the LIM-domain-only-2 transgenic and LN3 mouse models of T-ALL, le...
PubMed

Targeting cereblon in AML

Blood
The American Society of Hematology, 2021DOI: 10.1182/blood.2020009827© 2021 by The American Society of Hematology.
PubMed

Clonal cacophony of AML relapse

Blood
The American Society of Hematology, 2026DOI: 10.1182/blood.2025032092© 2026 American Society of Hematology. Published by Elsevier Inc. All rights are reserved, including those for text and data mining, AI training, and similar technologies.
PubMed

Predictors of clonal evolution and myeloid neoplasia following immunosuppressive therapy in severe aplastic anemia.

Leukemia
Predictors, genetic characteristics, and long-term outcomes of patients with SAA who clonally evolved after immunosuppressive therapy (IST) were assessed. SAA patients were treated with IST from 1989–2020. Clonal evolution was categorized as “hig...
PubMed

Exosomes and Extracellular Vesicles in Myeloid Neoplasia: The Multiple and Complex Roles Played by These “Magic Bullets”

Biology (Basel)
Multidisciplinary Digital Publishing Institute (MDPI), 2021DOI: 10.3390/biology10020105http://creativecommons.org/licenses/by/4.0/
SIMPLE SUMMARY: Extracellular vesicles (EVs) are released by the majority of cell types and can be isolated from both cell cultures and body fluids. They are involved in cell-to-cell communication and may shuttle different messages (RNA, DNA, and pro...
PubMed

PIKing the right target in AML

Blood
The American Society of Hematology, 2024DOI: 10.1182/blood.2024024035© 2024 American Society of Hematology. Published by Elsevier Inc. All rights are reserved, including those for text and data mining, AI training, and similar technologies.
PubMed

Context is key for FLT3-ITD

Blood
The American Society of Hematology, 2023DOI: 10.1182/blood.2022019135© 2023 by The American Society of Hematology.